Hepatitis C Virus-Induced ROS/JNK Signaling Pathway Activates the E3 Ubiquitin Ligase Itch to Promote the Release of HCV Particles via Polyubiquitylation of VPS4A | Journal of Virology
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Identification of a bilirubin receptor that may mediate a component of cholestatic itch | eLife
Hepatitis C Virus-Induced ROS/JNK Signaling Pathway Activates the E3 Ubiquitin Ligase Itch to Promote the Release of HCV Particles via Polyubiquitylation of VPS4A | Journal of Virology
High throughput screening for inhibitors of the HECT ubiquitin E3 ligase ITCH identifies antidepressant drugs as regulators of autophagy | Cell Death & Disease
Targeting TRP ion channels for itch relief | SpringerLink
The Ubiquitin Ligase Itch Regulates Apoptosis by Targeting Thioredoxin-interacting Protein for Ubiquitin-dependent Degradation - ScienceDirect
Hepatitis C Virus-Induced ROS/JNK Signaling Pathway Activates the E3 Ubiquitin Ligase Itch to Promote the Release of HCV Particles via Polyubiquitylation of VPS4A | Journal of Virology
ITCH as a potential therapeutic target in human cancers - ScienceDirect
Targeting TRP ion channels for itch relief | SpringerLink
ITCH as a potential therapeutic target in human cancers - ScienceDirect
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High throughput screening for inhibitors of the HECT ubiquitin E3 ligase ITCH identifies antidepressant drugs as regulators of autophagy | Cell Death & Disease
ITCH as a potential therapeutic target in human cancers. - Abstract - Europe PMC
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The Ubiquitin Ligase Itch Regulates Apoptosis by Targeting Thioredoxin-interacting Protein for Ubiquitin-dependent Degradation - ScienceDirect
The Ubiquitin Ligase Itch Regulates Apoptosis by Targeting Thioredoxin-interacting Protein for Ubiquitin-dependent Degradation - ScienceDirect
Identification of a bilirubin receptor that may mediate a component of cholestatic itch | eLife
Identification of a bilirubin receptor that may mediate a component of cholestatic itch | eLife
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Hepatitis C Virus-Induced ROS/JNK Signaling Pathway Activates the E3 Ubiquitin Ligase Itch to Promote the Release of HCV Particl
Astrocytic STAT3 activation and chronic itch require IP3R1/TRPC-dependent Ca2+ signals in mice - ScienceDirect
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